@genesdev.bsky.socialRESEARCH PAPER: Multimerization of ELAV in Drosophila is essential for directing neuronal alternative splicing and polyadenylation programs
By Dix, McQuarrie, Bräuer et al. and Matthias Soller
➡️ https://genesdev.cshlp.org/content/40/19-20/1669.abstract
@genesndiseases.bsky.social#COVID19 may leave a distinct brain signature.
This study links post-COVID brain changes to #olfactory-related regions and identifies S1 pyramidal #neuron activity as a key correlate of structural brain alterations.
#OpenAccess in #GenesAndDiseases: doi.org/10.1016/j.ge...
@kranhansya.bsky.socialロカロカダンス 踊ってみた #個人vtuber #Vtuber #アニメダンス #neuron
youtube.com/shorts/xxJ5r...
@talius2.bsky.socialTiny #polymer #devices mimic #neuron #firing, pointing toward #energyefficient #edgecomputing
MIT researchers engineered a compact computing platform combining processing and memory in one energy-efficient device, for adaptive next-gen electronics >> scitechupdates.com/t...
@wirepulleroo.bsky.socialWhy can binge eating feel compulsive? In a mouse model, high-fat-diet training altered an anterior zona incerta-to-VTA circuit linked to dopamine release. Fascinating, but not yet a treatment or direct human explanation. www.cell.com/neuron/fullt... #Neuroscience
@newsen.bsky.socialBiological Computing Co. Collaborates with AWS for Revolutionary Neuron-Derived AI Video Model #United_States #San_Francisco #AWS #Biological_Computing_Co #Neuron-Derived_AI
@proteinimaging.bsky.socialHere you can see a cryoEM structure of native GABA-A receptor from rat cerebella in complex with GABA (PDB code: 9OUP)
Rendering by Francisco J. Enguita made with #ProteinImager
#SciArt #molecularart #gaba #receptor #neuron #cortex #neurotransmitter #cryoem
@sesarju.bsky.social🙌🏼 #NEURON project has officially kicked off!
Funded by #SESARJU under #HorizonEurope, the project brings together 25 partners to develop new solutions for safer and more efficient airport operations through automation, #AI & digital technologies.
@wirepulleroo.bsky.social🧠 Low brain glucose may be more than a symptom of Tau disease. Hypometabolism and p-Tau synergize to trigger neuronal necroptosis via a p-Tau-RIPK1 hub. Restoring A20 or blocking the interaction eased brain atrophy in Tau mice. Preclinical, but striking.