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@zenhaus.bsky.socialSep 25, 2026, 3:29 PM

Zen Haus customers return for peace of mind 🕊️

They trust:

🌱 Premium non-GMO ingredients
🥽 Transparency
🌍 Eco-friendly practices
🥼 Products used in #HolisticHealth centers

Invest in a lifestyle of #wellness and integrity 🙏

amazon.com/zenhaus

#Iodine #Immune #VitaminD

@foodnourish.bsky.socialSep 25, 2026, 8:05 AM

📣 New Podcast! "Why Grandma’s classic cold remedy with Garlic actually works" on @Spreaker #allicin #alliinase #biohacking #colds #cooking #fitness #flu #food #garlic #healing #health #holistic #immune #immunity #nutrition #prevention #remedies #science #supplements #wellness

@cleeterichter.bsky.socialSep 23, 2026, 9:26 AM

We are looking for interested and motivated Masters or Medical students to help us investigate the immunometabolic changes in #immune cells during #cachexia. Apply today and join us at the lab of @andreasbergthaler.bsky.social and @meduniwien.ac.at

pii.meduniwien.ac.at/en/our-depar...

@toppnews.bsky.socialSep 23, 2026, 2:13 AM

#biotechnology #cancer #immune cells #research #Signal #Yang
🌐 www.newsmart.ch

@zenhaus.bsky.socialSep 21, 2026, 11:45 PM

Unleash the inner hero with #Glutathione! 💖

The master antioxidant is a secret weapon against daily stressors, helping you reclaim your vitality, glow and shine. 🥋

Ready to conquer your day? 📿

myzenhaus.com/glutathione

#Iodine #VitaminD #Immune #Supplements

amazon.com/zenhaus

woman meditating in grey-brown mitsy forest scene
@vanderbiltvicb.bsky.socialSep 21, 2026, 3:27 PM

How do colorectal tumors keep #immune cells out?

New #research reveals how DDR1-containing nanoparticles help create barriers that exclude immune cells, identifying a potential therapeutic #target.

Read: bit.ly/4AtOMte

More VICB publications: medschool.vanderbilt.edu/vicb/publica...

Scientific illustration comparing DDR1-low and DDR1-high tumors. DDR1-low tumors show disorganized collagen, less contractile fibroblasts, and CD8+ T cells entering the tumor. DDR1-high tumors release supermeres enriched in DDR1, increasing fibroblast contraction and collagen alignment to form a barrier that prevents CD8+ T cells from penetrating the tumor.
@allermd.bsky.socialSep 18, 2026, 4:14 PM

#Nasal #Epithelial #Immune #Signatures Are Associated With #Age-#Dependent #Asthma #Trajectories in Early Life. Open Access. ivancevichmd.blogspo...

#Nasal #Epithelial #Immune #Signatures Are Associated With #Age-#Dependent #Asthma #Trajectories in Early Life. Open Access. https://ivancevichmd.blogspot.com/2026/09/nasal-epithelial-immune-signatures-are.html#more
@plosbiology.orgSep 18, 2026, 1:01 PM

Most #bacterial #immune systems raise an alarm when they sense a #phage. This Primer explores a @plosbiology.org study showing how type II Panoptes reverses this logic by maintaining a quiet signal & sensing infection through its sudden disappearance 🧪 Paper: buff.ly/1ixwg9v Primer: buff.ly/WmnHZBp

Type II Panoptes inverts the logic of nucleotide signaling in bacterial immunity. Top: In type III CRISPR-Cas, Cas10 synthesizes cyclic oligoadenylates (cAn) only upon infection; the signal binds and activates a CARF effector, arresting growth. Type II Panoptes does the reverse. Its mCpol enzyme produces cA3 constitutively at low levels, and this signal keeps the CARF-TM effector repressed. Infection is sensed not as the appearance of a signal, but as its disappearance: when phage enzymes (Acb1 given as an example) deplete cA3, the effector is released and growth arrests. Bottom: Because Acb1 evolved to disable cyclic-nucleotide defenses such as type III CRISPR, a phage carrying it strips cA3 as collateral damage and triggers Panoptes. A phage lacking Acb1 avoids this trap but remains vulnerable to CRISPR. Together, the two systems could impose an evolutionary trade-off on the phage.
@talius2.bsky.socialSep 18, 2026, 6:31 AM

A single #IV treatment #retrains #immune cells to fight #autoimmune diseases in early trial

Researchers developed a revolutionary IV drip that delivers engineered viruses to reprogram immune cells in the body, eliminating complex CAR-T manufacturing
scitechupdates.com/a...

@plosbiology.orgSep 17, 2026, 12:55 PM

Acute #infections have been linked to #cancer progression, but they can also elicit #AntiTumor immune responses. This study explores gaps in understanding how infections differentially skew #immune programs to promote or restrain cancer progression @plosbiology.org 🧪 buff.ly/QfAWXBo

Upper left – healthy lung with a rare dormant cancer cell (green). Lower right – lung after infection with respiratory virus, showing expansion of dormant cancer cells (green cells; pink nucleus indicates active cell cycling). Image credit: Michael DeGregori.
@plosbiology.orgSep 17, 2026, 8:05 AM

Most #bacterial #immune systems raise an alarm when they sense a #phage. This Primer explores a @plosbiology.org study showing how type II Panoptes reverses this logic by maintaining a quiet signal & sensing infection through its sudden disappearance 🧪 Paper: buff.ly/1ixwg9v Primer: buff.ly/WmnHZBp

Type II Panoptes inverts the logic of nucleotide signaling in bacterial immunity. Top: In type III CRISPR-Cas, Cas10 synthesizes cyclic oligoadenylates (cAn) only upon infection; the signal binds and activates a CARF effector, arresting growth. Type II Panoptes does the reverse. Its mCpol enzyme produces cA3 constitutively at low levels, and this signal keeps the CARF-TM effector repressed. Infection is sensed not as the appearance of a signal, but as its disappearance: when phage enzymes (Acb1 given as an example) deplete cA3, the effector is released and growth arrests. Bottom: Because Acb1 evolved to disable cyclic-nucleotide defenses such as type III CRISPR, a phage carrying it strips cA3 as collateral damage and triggers Panoptes. A phage lacking Acb1 avoids this trap but remains vulnerable to CRISPR. Together, the two systems could impose an evolutionary trade-off on the phage.
@plosbiology.orgSep 17, 2026, 7:55 AM

R1-32-like public #antibodies drive #SARSCoV2 #virus to acquire #SpikeProtein mutations that promote #immune escape. This study characterises 4 such antibodies, showing that they acquire tolerance to SARS-CoV-2 antigenic drift via somatic hypermutation @plosbiology.org 🧪 buff.ly/iy4qUOe

Structures of mutation-tolerant antibodies in complex with SARS-CoV-2 BA.4/5 S-trimer and ACE2. Top: Cryo-EM densities of BA.4/5 S-R/6P S-trimer bound to Fab and ACE2 at 3:3:3 molar ratio. C092, C807, BD56-104, and BD56-597 Fabs, NTD, RBD, and ACE2 are colored in magenta, blue, cyan, and dark green, respectively; the rest of the S-trimer is colored in gray. The 3 RBDs in “up” positions are indicated, each simultaneously engaging both ACE2 and antibody. Bottom: Cryo-EM densities of BA.4/5 S1 bound to Fab and ACE2. C092, C807, BD56-104, and BD56-597 Fab-HC, Fab-LC, NTD, RBD, and ACE2 are colored in magenta, purple, blue, cyan, and dark green, respectively; the rest of the S1 is colored in gray.
@plosbiology.orgSep 16, 2026, 4:30 PM

Acute #infections have been recently linked to #cancer progression, but they can also elicit #AntiTumor immune responses. This study explores gaps in understanding how infections differentially skew #immune programs to promote or restrain cancer progression @plosbiology.org 🧪 buff.ly/QfAWXBo

Upper left – healthy lung with a rare dormant cancer cell (green). Lower right – lung after infection with respiratory virus, showing expansion of dormant cancer cells (green cells; pink nucleus indicates active cell cycling). Image credit: Michael DeGregori.
@plosbiology.orgSep 16, 2026, 4:25 PM

R1-32-like public #antibodies drive #SARSCoV2 #virus to acquire #SpikeProtein mutations that promote #immune escape. This study characterises four such antibodies, showing that they acquire tolerance to SARS-CoV-2 antigenic drift via somatic hypermutation @plosbiology.org 🧪 buff.ly/iy4qUOe

Structures of mutation-tolerant antibodies in complex with SARS-CoV-2 BA.4/5 S-trimer and ACE2. Top: Cryo-EM densities of BA.4/5 S-R/6P S-trimer bound to Fab and ACE2 at 3:3:3 molar ratio. C092, C807, BD56-104, and BD56-597 Fabs, NTD, RBD, and ACE2 are colored in magenta, blue, cyan, and dark green, respectively; the rest of the S-trimer is colored in gray. The 3 RBDs in “up” positions are indicated, each simultaneously engaging both ACE2 and antibody. Bottom: Cryo-EM densities of BA.4/5 S1 bound to Fab and ACE2. C092, C807, BD56-104, and BD56-597 Fab-HC, Fab-LC, NTD, RBD, and ACE2 are colored in magenta, purple, blue, cyan, and dark green, respectively; the rest of the S1 is colored in gray.
@plosbiology.orgSep 16, 2026, 12:47 PM

R1-32-like public #antibodies drive #SARSCoV2 #virus to acquire #SpikeProtein mutations that promote #immune escape. This study characterises four such antibodies, showing that they acquire tolerance to SARS-CoV-2 antigenic drift via somatic hypermutation @plosbiology.org 🧪 plos.io/3TcJrFA

Structures of mutation-tolerant antibodies in complex with SARS-CoV-2 BA.4/5 S-trimer and ACE2. Top: Cryo-EM densities of BA.4/5 S-R/6P S-trimer bound to Fab and ACE2 at 3:3:3 molar ratio. C092, C807, BD56-104, and BD56-597 Fabs, NTD, RBD, and ACE2 are colored in magenta, blue, cyan, and dark green, respectively; the rest of the S-trimer is colored in gray. The 3 RBDs in “up” positions are indicated, each simultaneously engaging both ACE2 and antibody. Bottom: Cryo-EM densities of BA.4/5 S1 bound to Fab and ACE2. C092, C807, BD56-104, and BD56-597 Fab-HC, Fab-LC, NTD, RBD, and ACE2 are colored in magenta, purple, blue, cyan, and dark green, respectively; the rest of the S1 is colored in gray.
@plosbiology.orgSep 16, 2026, 9:04 AM

Acute #infections have been recently linked to #cancer progression, but they can also elicit #AntiTumor immune responses. This article explores our gaps in understanding how infections differentially skew #immune programs to promote or restrain cancer progression @plosbiology.org 🧪 plos.io/3UYYcMS

Upper left – healthy lung with a rare dormant cancer cell (green). Lower right – lung after infection with respiratory virus, showing expansion of dormant cancer cells (green cells; pink nucleus indicates active cell cycling). Image credit: Michael DeGregori.
@yaleschoolofmed.bsky.socialSep 15, 2026, 2:54 PM

A YSM #study in Cell Press Blue investigates how #rhinoviruses (RVs), which cause the common cold and respiratory issues, trigger varying #immune responses in the nasal epithelium, the tissue lining the nose: medicine.yale.edu/publication-...

Antibodies bind to a rhinovirus.
@goetheuni.bsky.socialSep 15, 2026, 1:01 PM

🧪💥 What happens to B cells that could attack the body? | Christopher Goodnow showed that the #immune system does not always eliminate them, but places them into a safety mode. He will receive the 2027 Paul Ehrlich and Ludwig Darmstaedter Prize for his research. Congrats! 🔗 tinygu.de/pm-peld-2027...

@stillrollingoutdoors.comSep 15, 2026, 12:00 AM

#NervousSystem responds to environmental signals whether you're focused on it or not. 20 min. in nature drops #cortisol and shifts #immune function. Not because you did it correctly. Your body was waiting for the conditions.

buff.ly/cqzq5uH

#MentalHealth #Autoimmune #ChronicLife #GetOutside

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